The Heart, Punctured by Covid Vax Spike Proteins … Ouch! By Brian Simpson

A new study presented at the European Society of Cardiology Congress, has detailed the Covid-19 vaccine induced spike protein could change cells in the heart disrupting its regular function.  The spike protein binds to cells called pericytes, which in turn release chemicals causing an inflammatory response. The paper notes: “This mechanism has the potential to spread cellular and organ injury beyond the infection sites and may have important clinical implications.

For instance, in patients with disrupted endothelial barrier and increased vascular permeability due to underlying diseases, such as hypertension, diabetes, and severe obesity, S protein molecules could easily spread to the PC compartment and cause, or exacerbate, microvascular injury.”

“Blocking the CD147 receptor may help protect the vasculature of the most vulnerable patients from infection and the collateral damage caused by the S protein.”

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“According to a new study, the COVID-19 vaccine induced spike protein could change cells in your heart disrupting its regular function.

Researchers have found a concerning link between certain heart cells and the spike protein of SARS-CoV-2, the virus that causes COVID-19. the COVID-19 vaccine induced spike protein could change cells in your heart disrupting its regular function This spike protein appears to change cells in the small blood vessels around the heart, disrupting their regular function.

The pre-print study, was presented at the European Society of Cardiology Congress and shows that the spike protein binds to cells called pericytes.

These cells line the small vessels of the heart as well as other places around the human body. When the binding occurs, the pericytes begin to release chemicals that cause inflammation to the organ.

In the study, the team took small vessel cells from the heart and exposed them to the spike protein. The protein is used by the virus to attach itself to the cells.

Once the virus’s position is secured, the virus merges with the cell membrane, releasing its genetic material. This highjacks the cellular machinery, which begins replicating the virus, which then bursts out and spreads to other cells.

“This mechanism has the potential to spread cellular and organ injury beyond the infection sites and may have important clinical implications.

For instance, in patients with disrupted endothelial barrier and increased vascular permeability due to underlying diseases, such as hypertension, diabetes, and severe obesity, S protein molecules could easily spread to the PC compartment and cause, or exacerbate, microvascular injury,” the authors wrote in the paper.

“Blocking the CD147 receptor may help protect the vasculature of the most vulnerable patients from infection and the collateral damage caused by the S protein.”

If the spike protein alone is capable of affecting cells’ behavior, it’s concerning. It suggests that even cells that are not being infected can be harmed by the vaccine induced spike proteins.

The team found that by blocking the CD147 receptor on those cells, they reduce the effect of the spike protein on the pericytes, although there was still inflammation.

Pericytes are found all over the body, including the brain and central nervous system. If the mechanism can be stopped in patients it might reduce the complication and further investigations might produce better ways to stop the spike protein.”