Brush Your Teeth or Risk Your Heart: The Deadly Link Between Gum Disease and Health, By Mrs (Dr) Abigail Knight (Florida)
A ground-breaking study from Japanese researchers has uncovered a chilling truth: the bacteria lurking in your gums could be a silent killer, travelling through your bloodstream to scar your heart and trigger life-threatening irregular heartbeats. As reported by Natural News, Porphyromonas gingivalis, a common culprit behind gum disease, doesn't just rot your teeth, it can invade the heart's left atrium, causing fibrosis and increasing the risk of atrial fibrillation (A Fib), a condition linked to strokes and heart failure. This is more than a health warning; it's a call to take personal responsibility for your body in a world where the deep state's reckless biolabs, like those tampering with aerosolised Ebola, already threaten humanity's survival. Cleaning your teeth isn't just hygiene, it's a patriotic act to protect yourself and your country!
The study, published in Circulation and led by Hiroshima University's Shunsuke Miyauchi, reveals how P. gingivalis escapes infected gums, enters the bloodstream, and settles in the heart's left atrium. Once there, it triggers scarring (fibrosis) that disrupts electrical signals, leading to A Fib, a heart rhythm disorder affecting 60 million people globally, with cases nearly doubling from 2010 to 2019. In a study of 68 A Fib patients undergoing heart surgery, those with severe gum disease had higher levels of P. gingivalis in their heart tissue, directly correlating with worse scarring. Mouse experiments were even more alarming: infected mice were six times more likely to develop abnormal heart rhythms, with 30% showing A Fib after 18 weeks compared to just 5% of uninfected mice.
This isn't just a lab curiosity. Clinicians have long noticed that gum disease patients face higher heart risks, with a meta-analysis linking periodontitis to a 30% increased chance of A Fib. P. gingivalis is a stealth invader, hiding inside cells to evade immune defences and spiking biomarkers like galectin-3 and Tgfb1, which drive inflammation and scarring. As Miyauchi told News Medical, the bacteria "invades the circulatory system via periodontal lesions and translocates to the left atrium," where its presence worsens with the severity of gum disease.
For the MAGA movement, including Make Australia Great Again, this study hits home in multiple ways. First, it underscores personal responsibility, a core America/Australia First value. In an era where the deep state gambles with our lives in biolabs (like the Fort Detrick facility caught in a lover's spat that risked an Ebola outbreak discussed at the blog yesterday), you can't rely on the government to keep you safe … the opposite in fact. Brushing twice daily, flossing, and visiting the dentist aren't just chores, they're acts of defiance against a system that's failing us. As Dr. Jessica Pharar told The Epoch Times, bleeding gums signal gingivitis, and "bright red angry-looking gums" mean you're on the path to severe disease. Ignoring this invites P. gingivalis to wreak havoc, not just in your mouth but in your heart.
Second, this connects to the broader Western decline we fight against. France's "Mexicanisation" with crypto kidnappings shows what happens when elites let crime fester; America's biolab scandals reveal a similar rot in science. Gum disease, affecting 2 in 5 U.S. adults per Dr. Sandip Sachar, is another symptom of neglect, both personal and societal. Chronic gum infections are linked to diabetes, Alzheimer's, cancers, and even joint replacement failures, burdening our healthcare system and weakening Western nations.
MAGA sees health as national security. A strong America/Australia needs strong citizens, not a population crippled by preventable diseases. The Hiroshima study shows that gum disease isn't a minor issue, it's a gateway to systemic collapse, much like open borders or unchecked bioweapons research. The fact that P. gingivalis has been found in brains, livers, placentas, and arterial plaques underscores its threat. As Miyauchi's team pushes for medical-dental collaboration in Hiroshima, MAGA demands the same in America and Australia: integrate oral health into cardiovascular care, educate the public, and make dental checkups accessible, not a luxury.
This also exposes Big Pharma's failures. The US Senate hearing on biolabs, rife with "pharmaceutical talking points," reminds us that the same elites pushing vaccines and remdesivir ignore root causes like oral health. RFK Jr.'s presence at that hearing aligns with MAGA's push for truth over profit-driven science. Why isn't Big Pharma funding research to block P. gingivalis's heart invasion? Because there's no pill for prevention when brushing and flossing work better, and cost less.
The good news? You have the power to fight back. The Hiroshima study proves that regular oral hygiene can prevent and even reverse gum disease, potentially shielding your heart. Here's a plan to take control:
1.Commit to Daily Defence: Brush twice daily for two minutes, floss thoroughly, and use a probiotic mouthwash (never alcohol-based) to kill P. gingivalis. As Dr. Sachar advises, consistency is key to stopping gum inflammation before it spreads.
2.Get Regular Checkups: Schedule dental cleanings every six months. Early detection of gingivitis can stop P. gingivalis from entering your bloodstream. Demand affordable dental care, another MAGA fight against elitist healthcare systems.
3.Spread the Word: Share this study on X, tagging influencers like @HealthRanger or @EpochTimes. Educate your community about the gum-heart link. A healthy country starts with informed patriots.
4.Push for Policy Change: In America, support leaders like Trump and Rand Paul who challenge Big Pharma and reckless science; Australia, well that's a problem, but there are still good advocates like Senator Malcolm Roberts. Advocate for dental health in Medicare and Medicaid to protect vulnerable Americans/Australians from gum disease's fallout.
5.Live Nation First: A strong body fuels a strong nation. Eat clean, avoid sugar that feeds gum bacteria, and exercise to boost immunity. Personal health is a patriotic duty.
The Japanese study isn't just about teeth, it's a warning that small neglects can lead to catastrophic consequences, much like a punctured hazmat suit in a biolab or a kidnapping on a Paris street. MAGA stands for taking charge, whether it's securing borders, defunding dangerous labs, or cleaning your gums. Every brushstroke is a strike against the chaos threatening our nations. Let's make America and Oz healthy again, one smile at a time!
https://www.naturalnews.com/2025-05-16-gum-bacteria-invade-heart-cause-rhythm-problems.html
Japanese researchers discovered bacteria from gum infections can travel through the bloodstream to heart tissue, potentially causing dangerous irregular heartbeats.
The bacterium P. gingivalis can escape from infected gums to the left atrium of the heart, causing scarring that increases the risk of atrial fibrillation linked to stroke and heart failure.
Study of 68 patients with AFib found those with severe gum disease had higher levels of P. gingivalis in their heart tissue, corresponding with increased scarring.
Experiments on mice showed that P. gingivalis infection led to a sixfold increase in abnormal heart rhythms compared to uninfected mice.
Regular oral hygiene including twice-daily cleaning, flossing, and dental checkups can prevent and even reverse gum disease, potentially protecting both oral and cardiovascular health.
A silent killer may be lurking in your gums, according to a new study. Japanese researchers have discovered that bacteria responsible for common gum infections can travel through the bloodstream and directly invade heart tissue, potentially triggering dangerous irregular heartbeats.
The groundbreaking study, published in the journal Circulation, found that Porphyromonas gingivalis, a bacterium known to cause gum disease, can escape from infected gums and make its way to the left atrium of the heart. Once there, these oral invaders cause scarring of heart tissue, increasing the risk of atrial fibrillation (AFib), irregular heartbeats linked to higher stroke and heart failure risks.
Researchers from Hiroshima University examined heart tissue from 68 patients with AFib who had undergone heart surgery. They discovered that patients with severe gum disease had significantly higher levels of P. gingivalis bacteria in their heart tissue, which corresponded with increased heart scarring.
"The causal relationship between periodontitis and atrial fibrillation is still unknown, but the spread of periodontal bacteria through the bloodstream may connect these conditions," said study first author Shunsuke Miyauchi in a statement.
From mouth to heartTo confirm their findings, the research team created an experimental model using mice. When infected with P. gingivalis, the mice developed increased heart tissue scarring and were six times more likely to develop abnormal heart rhythms compared to uninfected mice.
The longer the infection persisted, the worse the outcomes became. After 18 weeks, 30% of infected mice developed atrial fibrillation when their hearts were electrically stimulated, compared to just 5% of uninfected mice.
Miyauchi explained that P. gingivalis "invades the circulatory system via the periodontal lesions and further translocates to the left atrium," noting that the bacterial presence directly correlated with the severity of periodontitis. Once established in the atrium, the bacteria "exacerbates atrial fibrosis, which results in higher AFib inducibility."
These findings provide a potential explanation for something cardiologists and dentists have observed for years: people with gum disease have higher rates of heart problems. A recent meta-analysis linked gum disease to a 30% higher chance of developing AFib.
Warning signs and natural preventionAny bleeding in the gums indicates gingivitis, according to dentist Dr. Jessica Pharar. "If you see bright red angry-looking gums, that is a good indication you are progressing more severe gingivitis," she told The Epoch Times.
The severity of gum inflammation, not just the size of gum pockets, was strongly associated with bacterial invasion of heart tissue. This suggests that active inflammation creates opportunities for bacteria to escape into the bloodstream.
The good news? Natural approaches can effectively prevent and even reverse gum disease.
"Cleaning your mouth twice a day is imperative. Regular check ups at the dentist are essential to protect your overall dental health," Pharar advised.
Dr. Sandip Sachar, a dentist in New York City, emphasized that gum disease remains "an important public health problem in the United States as 2 in 5 adults are affected by some form of this disease." He recommended regular brushing, flossing, and professional dental cleanings.
The implications extend beyond heart health. Research has linked chronic gum infections to diabetes, Alzheimer's disease, certain cancers, and even joint replacement infections.
As researchers continue investigating the specific mechanisms by which P. gingivalis affects heart cells, this study highlights an often-overlooked connection between oral and cardiovascular health. Simple daily habits like proper brushing and flossing may be powerful natural defenses against serious heart conditions, proving that protecting your smile could also be saving your heart."
Tempted to skip the floss? Your heart might thank you if you don't. A new study from Hiroshima University (HU) finds that the gum disease bacterium Porphyromonas gingivalis (P. gingivalis) can slip into the bloodstream and infiltrate the heart. There, it quietly drives scar tissue buildup-known as fibrosis-distorting the heart's architecture, interfering with electrical signals, and raising the risk of atrial fibrillation (AFib).
Clinicians have long noticed that people with periodontitis, a common form of gum disease, seem more prone to cardiovascular problems. One recent meta-analysis has linked it to a 30% higher risk of developing AFib, a potentially serious heart rhythm disorder that can lead to stroke, heart failure, and other life-threatening complications. Globally, AFib cases nearly doubled in under a decade, rising from 33.5 million in 2010 to roughly 60 million by 2019. Now, scientific curiosity is mounting in how gum disease might be contributing to that surge.
Past research has pointed to inflammation as the likely culprit. When immune cells in the gums rally to fight infection, chemical signals they release can inadvertently seep into the bloodstream, fueling systemic inflammation that may damage organs far from the mouth.
But inflammation isn't the only threat escaping inflamed gums. Researchers have discovered DNA from harmful oral bacteria in heart muscle, valves, and even fatty arterial plaques. Among them, P. gingivalis has drawn particular scrutiny for its suspected role in a growing list of systemic diseases, including Alzheimer's, diabetes, and certain cancers. It has previously been detected in the brain, liver, and placenta. But how it manages to take hold in the heart has been unclear. This study, published in Circulation, provides the first clear evidence that P. gingivalis in the gums can worm its way into the left atrium in both animal models and humans, pointing to a potential microbial pathway linking periodontitis to AFib.
The causal relationship between periodontitis and atrial fibrillation is still unknown, but the spread of periodontal bacteria through the bloodstream may connect these conditions."
Shunsuke Miyauchi, study first author, assistant professor at HU's Graduate School of Biomedical and Health Sciences
"Among various periodontal bacteria, P. gingivalis is highly pathogenic to periodontitis and some systemic diseases outside the oral cavity. In this study, we have addressed these two key questions: Does P. gingivalis translocate to the left atrium from the periodontitis lesion? And if so, does it induce the progression of atrial fibrosis and AFib?"
Examining the gum disease-AFib linkTo simulate how P. gingivalis might escape the mouth and wreak havoc elsewhere, researchers created a mouse model using the bacterium's aggressive W83 strain. They divided 13-week-old male mice into two groups: one had the strain introduced into the tooth pulp, the other remained uninfected. Each was further split into subgroups and observed for either 12 or 18 weeks to track the cardiovascular risks of prolonged exposure.
Intracardiac stimulation-a diagnostic technique for arrhythmia-revealed no difference in AFib risk between infected and uninfected mice at 12 weeks. But by week 18, tests showed that mice exposed to the bacterium were six times more likely to develop abnormal heart rhythms, with a 30% AFib inducibility rate compared to just 5% in the control group.
To see if their model accurately replicated periodontitis, the researchers examined jaw lesions and found its telltale signs. They detected tooth pulp decay and microabscesses caused by P. gingivalis. But the damage did not stop there. They also spotted the bacterium in the heart's left atrium, where infected tissue had turned stiff and fibrous. Using loop-mediated isothermal amplification to detect specific genetic signatures, the team confirmed that the P. gingivalis strain they had introduced was present in the heart. In contrast, the uninfected mice had healthy teeth and no trace of the bacterium in heart tissue samples.
Twelve weeks after infection, mice exposed to P. gingivalis already showed more heart scarring than their uninfected counterparts. At 18 weeks, scarring in the infected mice had climbed to 21.9% compared to the likely aging-related 16.3% in the control group, suggesting that P. gingivalis may not just trigger early heart damage, but also speed it up over time.
And this troubling connection was not only seen in mice. In a separate human study, researchers analyzed left atrial tissue from 68 AFib patients who underwent heart surgery. P. gingivalis was found there, too, and in greater amounts in people with severe gum disease.
Past studies have shown that P. gingivalis can invade host cells and evade destruction by autophagosomes, the cellular garbage crew. This ability to hide inside cells suggests a way by which it can slip past immune defenses and trigger just enough inflammation to cause harm without being flushed out. Infected mice showed a spike in galectin-3, a biomarker for fibrosis, and higher expression of Tgfb1, a gene tied to inflammation and scarring.
The findings suggest that brushing, flossing, and regular dental checkups might do more than promote oral hygiene, they could also help protect the heart. Keeping gums healthy could block the gateway for a P. gingivalis invasion.
"P. gingivalis invades the circulatory system via the periodontal lesions and further translocates to the left atrium, where its bacterial load correlates with the clinical severity of periodontitis. Once in the atrium, it exacerbates atrial fibrosis, which results in higher AFib inducibility," Miyauchi said. "Therefore, periodontal treatment, which can block the gateway of P. gingivalis translocation, may play an important role in AFib prevention and treatment."
The team is now working to strengthen interdisciplinary collaboration between medical and dental professionals in Hiroshima Prefecture to improve cardiovascular care.
"For the next step, we're investigating the specific mechanisms by which P. gingivalis affects atrial cardiomyocytes," Miyauchi said. "We're also now focusing on establishing a collaborative medical and dental system in Hiroshima Prefecture to treat cardiovascular diseases, including atrial fibrillation. We aim to expand this initiative nationwide in the future."
Other co-authors in the study include HU School of Dentistry's Miki Kawada-Matsuo and Hitoshi Komatsuzawa from the Department of Bacteriology, Hisako Furusho, Ayako Nakajima, Pham Trong Phat, Masae Kitagawa, and Mutsumi Miyauchi from the Department of Oral and Maxillofacial Pathobiology, and Kazuhisa Ouhara from the Department of Periodontal Medicine; Hiroshima University Hospital's Hiromi Nishi and Hiroyuki Kawaguchi from the Department of General Dentistry, Noboru Oda, Takehito Tokuyama, Yousaku Okubo, Sho Okamura, and Yukiko Nakano from the Department of Cardiovascular Medicine, and Toru Hiyama from the Division of General Medicine; HU Collaborative Research Laboratory of Oral Inflammation Regulation's Fumie Shiba; and HU School of Medicine's Taiichi Takasaki and Shinya Takahashi from the Department of Surgery.
Source:
Journal reference:
Miyauchi, S., et al. (2025). Atrial Translocation of Porphyromonas gingivalis Exacerbates Atrial Fibrosis and Atrial Fibrillation. Circulation. doi.org/10.1161/circulationaha.124.071310.
Comments