A New Covid Vax Related Disease: VEXAS Syndrome By Chris Knight (Florida)

 VEXAS syndrome is an auto immune disorder that previously mainly infected males over 50 years. It results in inflammation, fever, and blood disorders that can severely damage organs. The disease has a high death rate, killing up to half of afflicted people within five year. The syndrome has a genetic basis, but is not inherited, with a mutation occurring on a gene in the X chromosome, so it was not thought that this syndrome could occur as an adverse effect of a vaccine.

Yet, a recent paper, the link at the end of this article, has shown cases of VEXAS syndrome arising after the Covid mRNA vax. A gloss on the highly technical details is given by Natural News.com, below. The interesting question is what mechanism leads to the mutation of the gene responsible for VEXAS? It must indicate that the mRNA spike protein is interacting with the DNA on the X chromosome, and causing the mutation.

If so, that is a whole new level of concern, indicating interaction with cellular DNA. Recall that these "safe and effective" vaxxes were supposed to just stay at the injection site, working their "magic," which turns out now to be black magic indeed.

https://www.naturalnews.com/2024-01-18-fully-vaccinated-covid-new-disease-vexas-syndrome.html

"People who were injected with "vaccines" for the Wuhan coronavirus (COVID-19) are increasingly being diagnosed with a new type of disease they are calling VEXAS syndrome, an autoinflammatory ailment that was first discovered in 2020 around the time Operation Warp Speed was launched by the Trump regime.

VEXAS syndrome, short for vacuoles, E1 enzyme, X-linked, autoinflammatory, somatic syndrome, is said to be caused by mutations in the innate immune cells, as well as a somatic mutation in the UBA1 gene found on the X chromosome. Most inflammatory diseases, by the way, are caused by dysfunction that arises in adaptive immune cells.

"Somatic mutations cannot be inherited, meaning individuals acquire this mutation later in life," explains The Epoch Times about the disease. "The mutation affects the stem cells in the bone marrow. The cells mature into specialized immune cells that circulate within the bloodstream."

"Immune cells carrying the UBA1 mutation are highly inflammatory, and once enough of them accumulate, patients start developing symptoms."

Autoinflammation caused by COVID jabs

Back in April, French scientists reported on the case of a 76-year-old man who almost immediately after getting jabbed for COVID with Pfizer's mRNA (modRNA) variety was diagnosed with VEXAS syndrome. His symptoms included tender bumps under the skin, rashes and purple spots on his limbs.

Skin problems are commonly reported among VEXAS patients, and the man was no exception. He was later determined by specialists to have the UBA1 mutation inherent to the disease.

"The rare incidence of VEXAS syndrome and the short delay of 3 days between vaccination and onset of symptoms were very suggestive of the vaccine's role as a trigger," the study authors, from Drôme Nord Hospitals, wrote.

Another patient, 72, developed similar symptoms, as well as a fever, fatigue, a cough and deep vein thrombosis. He was initially misdiagnosed with "long COVID," only to later also show evidence of the same UBA1 mutation as the first patient.

"In my experience, it is unlikely that VEXAS syndrome could have been triggered by an infection or COVID-19 vaccination," commented Dr. Sinisa Savic, an immunologist and associate clinical professor at the University of Leeds. "We know that as people age, they develop all sorts of mutations in the bone marrow ... That is why VEXAS is largely found in the elderly population."

Typically, VEXAS syndrome occurs in men over the age of 50. Both infections and vaccinations can trigger or worsen symptoms in people who are already on track to develop VEXAS syndrome.

"Anything that triggers an immune response can cause temporary worsening symptoms," Dr. Savic added. "I don't think there's any particular argument about that."

Among specialized immune cells, only innate immune cells have been found to carry the UBA1 mutation. Adaptive immune cells, meanwhile, form what is known as the "third" or last line of defense against disease, and these cells have not been found to carry the UBA1 mutation.

Dr. Savic believes that adaptive immune cells, including T and B cells, probably cannot survive long enough to become specialized if they carry the UBA1 mutation. Specialization of innate immune cells, conversely, appear to be less affected by the UBA1 mutation.

Both infections and vaccinations trigger responses in the immune system that are supposed to (in theory for vaccinations, anyway) form immune memory. This process does not occur in people with autoinflammatory conditions – in fact, an immune reaction can cause an imbalance that actually worsens a patient's condition.

"This is the case with any autoimmune or inflammatory condition because the immune system tries to control itself, but if you're then challenged by something else, then that level of control may be reduced," Dr. Savic said."